Back on the track after a long hiatus. Beautiful weather, 68º sunny. I ran my first 300m today on the new track and followed up with a 200m. Not all out but at a brisk pace. Both sprints, I predictably faded at the end, but it's a good start. My foot was feeling better, and would have felt much better if I hadn't hiked yesterday on some rocky trails. Couldn't resist the beautiful weather.
Hoka trainers on
stretches, drills, 100m, 60m, bands
Hoka rocket x2 trainers on
50m stride ~ 7.2
300m - 46.34. (14.44, 15.30, 16.60)
200m - 28.40 (13.48, 14.92)
I wanted to do more but I waited a bit too long after the 200m while talking with the track coach. HR hit 181 at the end of the 300m, 174 in the 200m. Have noticed an interesting phenomenon in regard to HR and breathing. Sometimes during recovery, my HR can drop below 130 while I'm still breathing hard. I remember times on the bike when my HR was 160 and I wasn't breathing hard at all. Don't know what this means.
Anyway, as I build up, maybe next time I can try 2x300m w/ 3min rest and aim for avg of 50 or so. My 46sec 300m might have been faster since I seemed to have drifted to the outside lane on the turn. Again, first time running turns on this track and the setting sun was in my face. This would equate to a mid 60s 400m pace. I think I could run about a 64 right now. Long time to go before outdoor Nationals. I hope they pin down a date by Xmas.
140.9 lbs after workout. Now that Roya is gone, I can be a bit more conscientious, both in my training in music and athletics. Still planning a bike climb Sunday with Bill.
The heart has significantly different stresses on it in sprinting vs steady state exercise, which is why sprinters and aerobic athletes hearts are different. Sprinting or strength sports develops thicker heart muscle walls which leads to reduced stroke volume and higher heart rate during exercise and recovery. Rarely this results in SCD. Aerobic athletes are at risk of developing arrhythmia which can be deadly, still rare but more frequent than the type of hypertrophy SCD sprinters can develop. Even at the same or lower heart rate the stresses of a sprint are significantly greater on the heart than steady state exercise mainly related to hemodynamics in the form of significantly higher peak exercise blood pressure which stimulates the heart adaptation necessary for peak sprint performance. So the outlier data you have for maximum heart rate and have slow heart rate recovery is probably a result of how you remodeled your heart. Most likely benign but it may be a sign. Your doctors will not be interested as it has weak predictive capability. The only way to know if it is pathological is with echo cardiogram where they can measure the thickness of your walls. Ideally at rest and at stress as they can look for abnormal wall motion. But medicine doesn't look for outlier problems they are ok with a few percent falling through the cracks. Sucks for the few percent. They won't even do a simple ECG. Ridiculous.
ReplyDeleteOff the soap box. Respiration plays a role in reducing acidosis. Sprint training induces acidosis and a compensatory mechanism is gas exchange through the lungs. So that is the likely explanation. It can be measured with partial gas analysis. Ability to buffer acidosis can be improved through training which is another reason why I was promoting off season non running progressive interval training. Acidosis is part of the reason you have trouble tolerating sprinting when you resume intense track training.
Can you find one example of SCD in a sprint race? I've not been able to find a single example. Usually happens with large football players, distance runners. After a second Agatston zero score, insurance won't likely pay for more detailed testing, which does suck. I will more likely die of cancer or some other disease than CVD.
DeleteI cannot. The incidence rate of any sport related SCD ranges from 1:40000. My opinion is if there were as many old people who practiced intense sprinting for decades as there are old aerobic athletes there would be examples to find. I bet there are 1000x more old cyclists, XC skiers , and swimmer master athletes than 400m sprinters.
DeleteThat is ranges from 1:40000 to 1:250000.
ReplyDeleteAgree with your assessment. But it is proven that up to 10 percent of zero CAC scores have non calcified plaques. So the risk, albeit low, is if you have some un calcified non visible plaque that ruptures due to high stress (which is my motivation for statins - pin them in place prior to ski season). I brought up sprinting as a possible contributor based on the fail model - which is that un calcified plaques are unstable and can rupture under stress, and that hemodynamic stress is actually greater in sprinting than steady state exertion. Irregardless of examples or lack thereof, those are facts so the model should be valid. On that note, it is better not to lay down or sit between intense intervals. For the stress ECG/echo they measure everything - my BP was 190/81 at exhaustion, which apparently is normal for a max effort, but they immediately have you lay down to capture heart wall motion with echo and BP jumped up to 230/90. That is the only point where I felt dizzy. So I can imagine that that kind stress could trigger a rupture - which is why they typically have you stop at the calculated 85 percent heart rate for your age. Can’t imagine stopping at 133 seems like a waste of time and would miss potential problems. Anyway I learned that remaining upright and moving is less hemodymically stressful- so less chance of plaque rupture. So keep moving after hard intervals. Since intense training, in one form or another, is a lifetime commitment, I previously stated to “work towards” ECG and echocardiogram over the next couple of years. If you get it while you are healthy it is valuable as a baseline if, heaven forbid, issues arise later. Of course it is also incredibly cool data and would be interesting to know how your heart has adapted to training. Nothing urgent, unless you are dizzy after intense intervals or see weird heart rate data that could indicate arrhythmias. Or if brother has arrhythmias or something weird on echocardiogram. Or if you notice irregular heart beats. There can be lots of reasons to have ECG or echocardiogram, to rule out chest pain (heartburn), skipped or irregular heartbeats, dizziness with intense exercise, etc..
ReplyDeleteI was told that's how Dan M. died, in a chair just after a hard bike TT.
DeleteOn the subject of heart rate increasing after finishing an interval. Or just not reducing quickly. Some watches have a more noticeable lag in readings I notice that particularly with my fitbit compared to an old Garmin.
ReplyDelete